RT Journal
A1 WALLGREN A
T1 PAthogenesis of acute serofibrinous pleurisy
JF American Journal of Diseases of Children
JO American Journal of Diseases of Children
YR 1929
FD October 1
VO 38
IS 4
SP 829
OP 833
DO 10.1001/archpedi.1929.01930100149017
UL http://dx.doi.org/10.1001/archpedi.1929.01930100149017
AB Acute serofibrinous pleurisy is now almost invariably regarded as due to tuberculosis. In what manner tuberculosis gives rise to it, is, on the other hand, somewhat obscure. As a rule, one has been content to state that the tuberculous infection in some manner produces the exudation. The most obvious and common explanation is that a subpleural tuberculous focus has provoked an irritation of the pleura.That a local tuberculous focus in the lung or the bronchial glands constitutes an important factor in the genesis of pleurisy is indicated by the fact that a focus of this kind can be detected on the side on which the exudation subsequently arises. If an opportunity presents itself of examining a child a short time before it contracts pleurisy, there are generally found homolateral hilar changes or a homolateral primary focus. Of fifty children examined before the development of pleurisy, forty-eight showed homolateral changes