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Sir.—I read with interest in the Journal (133:636-638, 1979) the article by Robson, Arbus, and Balfe, "Bartter's Syndrome: Differentiation Into Two Clinical Groups"; it confirms the findings I published with David et al in 1976.1 In addition, I observed that the infusion of angiotensin II produced an increase in urinary sodium excretion only in the patients with sodium wasting and that this renal sodium wastage occurred despite an increased aldosterone secretion.
I could not confirm some points