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III. The Georgia Epidemic

HENRY R. SHINEFIELD, MD; MARVIN BORIS, MD; JOHN C. RIBBLE, MD; ELLSWORTH F. CALE, MD; HEINZ F. EICHENWALD, MD
Am J Dis Child. 1963;105(6):663-673. doi:10.1001/archpedi.1963.02080040665017.
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Definition of Terms.—A glossary of terms used in this publication is included in a previous paper.1

In the preceding papers of this series, evidence has been presented which indicates that the 502A strain of Staphylococcus aureus is capable of colonizing the cord and nasal mucosa of newborn infants,1 and that the presence of this organism actively interferes with the subsequent acquisition of other coagulase positive staphylococci, including type 80/81, under epidemic conditions.2 The present communication presents additional evidence bearing on this point, as well as certain other data on the ecology of staphylococcal infection in newborn infants and their contacts.

During September and October of 1961, an increase in the rate of impetigo among infants discharged from a proprietary Atlanta hospital was noted by the pediatricians in the area. Preliminary studies were carried out by a group of investigators from the Communicable Disease Center between Nov

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