Capillary hemangiomas are benign tumors composed of capillary endothelial cells. Fewer than 25% of lesions are present at birth, but rapid development is common in the first months of life.3 Rapid proliferation of these lesions can produce ulceration, bleeding, infection, necrosis, and compression of adjacent structures.7 Large lesions may result in thrombocytopenia secondary to intralesional platelet sequestration (Kasabach-Merrit syndrome).7,8 Most capillary hemangiomas resolve spontaneously. Typically, 50% of capillary hemangioma resolve spontaneously by age 5 years, 70% by age 7 years, 90% by age 9 years, and the rest by adolescence.2,3 The clinical course of capillary hemangioma is similar for lesions from various sites. Since spontaneous regression is the rule, treatment is only indicated if visual disturbance is present.4,7 Corticosteroids are the primary pharmacologic treatment used to control hemangiomas.4,9 Interferon is effective in refractory cases.4 Use of intralesional cryotherapy and sclerosing agents are of limited value. Radiation treatment is only considered for life-threatening lesions. Because orbital capillary hemangiomas are highly vascular lesions with the blood supply directly from the internal or external carotid artery, surgical intervention poses considerable risk for massive blood loss and is rarely indicated.