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Am J Dis Child. 1925;29(5):603-610. doi:10.1001/archpedi.1925.04120290024004.
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All through the extensive studies of recent years in rickets, little has been done to investigate craniotabes. In the past, some investigators, notably Kassowitz1 and later Wieland,2 concerned themselves with the etiology and pathology of this clinical condition. They found it frequently associated with rickets, and the former believed that craniotabes was a manifestation of rickets and that all rickets, therefore, was congenital in origin. Wieland,3 Schloss4 and Finkelstein5 believed that the early congenital softenings were physiologic in nature and soon disappeared, while new softenings appearing on the hard bone later were "rachitic." Czerny6 states that craniotabes makes a positive diagnosis of rickets. Marfan7 says that true craniotabes is of rachitic origin, but believes syphilis to be the most common cause of craniotabes. De Stefano8 states that craniotabes is a manifestation either of rickets or of inherited syphilis. Thus, we see that


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